found the following online,.....not sure if u can understand all of this, but here u go :
Hemangiomas are classified pathologically by the predominant type of vascular channel (capillary, cavernous, arteriovenous, or venous) seen at histologic examination (Figure 1). Nonvascular components can also be seen in angiomatous lesions, particularly cavernous hemangioma, including fat, smooth muscle, fibrous tissue, bone, hemosiderin, and thrombus. Overgrowth of adipose tissue is most frequently associated with hemangiomas, and this characteristic led some authors in the past to refer to these lesions as angiolipomas. However, fat overgrowth should be considered a reactive phenomenon as opposed to a true neoplastic component; therefore, the term angiolipoma is not appropriate for the vast majority of these musculoskeletal vascular lesions (1,2,3). True angiolipomas are rare lesions of the subcutaneous tissue, most commonly in the forearm, and imaging of this neoplasm has not been reported to the best of our knowledge
The majority of hemangiomas that involve bone are discovered incidentally in asymptomatic patients. Men are affected twice as often as women, and lesions are usually discovered in the 4th 5th decades of life. Soft-tissue components may also be associated with these lesions. Osseous hemangioma is particularly common in the spine and calvaria and less frequently affects long bones such as the tibia, femur, and humerus.
Vertebral hemangioma is extraordinarily common, seen in 11% of the cases in one large autopsy series (10). It accounts for 28% of all skeletal hemangiomas (11). These lesions can involve only a portion of or the entire vertebral body and are multiple in one-third of the cases (12). The thoracic spine is the most common location for vertebral hemangiomas (Figure 2) (5,10,11,12).
At radiography, vertebral hemangiomas classically have a coarse, vertical, trabecular pattern, with osseous reinforcement (trabecular thickening) adjacent to the vascular channels that have caused bone resorption (13). This appearance on radiographs represents a response to stress and has been likened to corduroy. Vertebral fractures at the site of these hemangiomas are unusual because of this trabecular reinforcement. At CT, the thickened trabeculae are seen in cross section as small punctate areas of sclerosis, often called the polka-dot appearance. At MR imaging, areas of trabecular thickening have low signal intensity, regardless of the pulse sequence used (Figure 3). On T1-weighted MR images, the signal intensity of vertebral hemangiomas varies from low to high, depending on the degree of adipose tissue present (Figure 3). T2-weighted MR images usually show areas of very high intensity corresponding to the vascular components (14). CT or MR images obtained after intravenous administration of contrast material demonstrate lesion enhancement.
Vertebral hemangiomas occasionally cause neurologic symptoms from spinal cord compression, particularly if these lesions extend into the posterior elements or surrounding soft tissues, expand bone, or fractureAu: this seems to contradict the preceding paragraph, in which you indicated that fracture was unusual; please address discrepancy* (Figure 4) (5,14,15). The larger the degree of fat overgrowth in the stroma between thickened trabeculae (seen on CT images as low attenuation between thickened trabeculae and as areas of high intensity on T1-weighted images and intermediate intensity on T2-weighted images), the less likely these lesions will be symptomatic (inactive hemangioma) as shown by Laredo and coworkers (15).
Calvarial hemangiomas account for 20% of all hemangiomas and are most frequent in the frontal or parietal region (11). These lesions arise in the diploic space and cause expansion that often involves the outer table to a greater extent. At radiography and CT, a calvarial hemangioma commonly appears as a lytic lesion with a pattern of radiating, weblike or spoke-wheel, trabecular thickening (Figure 5) (5,10,11). This characteristic appearance, as in vertebral lesions, is caused by preexisting trabeculae that have become thickened through intramembranous bone formation adjacent to the angiomatous channels. Recognition of the pattern should alert the radiologist to the vascular nature of the lesion.
Osseous hemangiomas in other locations may also have radiating trabecular thickening on radiographs. Another common pattern is a bubbly bone lysis that creates a honeycomb, latticelike, or "hole-within-hole" appearance. These lytic areas are invariably multifocal and usually metaphyseal or epiphyseal. Bone lysis can have linear and circular components on radiographs, suggestive of a vascular lesion, with linear and circular elements representing vascular channels seen longitudinally and en face, respectively. However, these serpentine vascular channels are recognized more easily with CT and MR imaging. Characteristically, these channels have low signal intensity on T1-weighted images and very high signal intensity on T2-weighted images because of slow blood flow. In arteriovenous lesions with faster blood flow, low signal intensity may persist with all MR imaging pulse sequences. The appearance of osseous hemangiomas at bone and red blood cell labeled scintigraphy is variable, from photopenia to moderate increased activity (16,17).
Periosteal or cortical hemangiomas occur most frequently in the anterior tibial diaphysis. These lytic cortical lesions may also show the characteristic multifocal vascular channels (Figure 6) or be seen as a larger, nonspecific region of bone destruction. Cortical hemangiomas may predispose the bone to fracture, and periosteal reaction may accompany these lesions.
U r welcome...there was alot of info there and I have a HA ...so I am sorry I could not rewrite into my own words.....
That's ok. You do so much for so many, take care of yourself :)
I hope you feel better!