Thanks.
The hr also crossed my mind as I do run a low heart rate. 50s during day and 30s while asleep. Doc says i have a large bell curve as that when i exercise i can get all the way up to 180s.
But since i monitor my hr, I still get prematures with rates in 60s,70s,and even with exercise.
Another correlation ive put together is body position. If sot down i have more and if i go from standing to laying down quickly i will go into bigeminy for about a minute. Makes me think something with blood flow or para/sympathetic nervous system influence.
I get to actually get a coronary ct angio scan recommended by my old cardiologists after finding out my maternal grandfather died of sca but was from small coronary arteries than normal. I took alot of traits from him as being tall and active. My mother also has afib but was told from thyroid issues.
The alcohol thing is interesting. Doesnt make sense unless 1.5 mg of clonazepam is a low dose
There are two possible explainations (in fact three, but the calming effect is ruled out since you don't get the same effect from clonazepam. They are both GABA-A receptor agonists, they increase the effect of the inhibitory neurotransmitter - but alcohol does more than that).
The most likely:
Alcohol increases heart rate, which makes premature beats less likely to occur. In fact, in some rare cases, PVCs/PACs are treated with a pacemaker to increase heart rate to eliminate them.
The less likely:
Alcohol is a sodium/potassium channel blocker (class I/III antiarrhythmic properties). It prolongs QRS and QT intervals some. Which is why people with prolonged QT time shouldn't drink alcohol. This effect may reduce (or in some people increase) ectopics.