Aa
Please interpret test result
Hep b surface Antigen - Positive
Hep b sur Antibody - Negative
e- antibody- Reactive
Virus Core Igm : Negative
ALT -27
AST - 25
Virus DNA:
DNA Blood QL PCR : 13,600
Virus DNA SER QNA : 30, 738.
What treatment suggestions if any is recommended?
Hep b sur Antibody - Negative
e- antibody- Reactive
Virus Core Igm : Negative
ALT -27
AST - 25
Virus DNA:
DNA Blood QL PCR : 13,600
Virus DNA SER QNA : 30, 738.
What treatment suggestions if any is recommended?
Thank you for all the iputs. I'm glad to have this info before the Gastro. appt. I 'll see if I can get a referral to a a hepatologist as well. Will keep you posted.
Thanks again.
Thanks again.
And e-negatives shouldn't wait to see the viral load in the millions.
Also, the VAST majority of HCC is found in cirrhotic livers.
Also, the VAST majority of HCC is found in cirrhotic livers.
thats actually very true, zelly
percentages of hepatoma are found more in people with hbeag negative, and normal alt/ast.
doesnt mean this is a given and it will happen to you or me but it means that the fact that you're low in V/L count with normal ALT/AST doesnt gurantee you safety..
percentages of hepatoma are found more in people with hbeag negative, and normal alt/ast.
doesnt mean this is a given and it will happen to you or me but it means that the fact that you're low in V/L count with normal ALT/AST doesnt gurantee you safety..
Keep in mind that in the mutations the viral loads do not tend to go as high but can be doing significant damage. You probably won't see viral loads in the millions.
im confused.. what is the V/L count. chic mentioned 13,600 IU/ml and also 1300 IU/ml
well , if 13,600 IU/ml and normal ALT/AST then 2007 guideline suggests treatment ( thats going by the book )
if 1300 IU/ml then only monitoring is needed .
now having said that, please be aware that if you decide to go with treatment ten you are stuck with it for life. you will be treated indifently . the immeidate but not ultimate goal of treatment is for carrier with hbeag postive to convert into hbeag negative..then treatment may be continued after that for another 6-12 month and then we quit meds with close monitoring at first to make sure virus is contained kinda.
on the other hand, when your body had already seroconverted on its own ut the virus found away to mutate and activate itself then a treatment may in needed . you are aldready hbeag negative so you need to ask yourself what the reason for treatment is.. e postives want to turn negative but you're already negative .. so there's no end to treatment. you will have to go on it for life.
its a little hard to be in your shoes now.. you are above the 2000 IU/ml limit for no treatment but you're not shooting up in the millions either so you have a decision to make. if i were you and thats a only a personal opinion, i would monitor for a little bit of time.. one year mayb ...even two years.. 2 years still arent big of a time for monitoring considering if you choose to go with meds then its a life time commitment.. monitor closely for a year or two..maybe once every 3 months to see th elevel of your DNA.. if it goes lower then maybe you'll be ok without meds.. if you goes higher then maybe you needto settle with rlity an go on meds .
up to you and as i always say doctors know better.. but i would recommend you make an appointment with a hepatologist for a second opinion.. they are much more expensive but they do know one or two more things than a gastronologist..
good luck
well , if 13,600 IU/ml and normal ALT/AST then 2007 guideline suggests treatment ( thats going by the book )
if 1300 IU/ml then only monitoring is needed .
now having said that, please be aware that if you decide to go with treatment ten you are stuck with it for life. you will be treated indifently . the immeidate but not ultimate goal of treatment is for carrier with hbeag postive to convert into hbeag negative..then treatment may be continued after that for another 6-12 month and then we quit meds with close monitoring at first to make sure virus is contained kinda.
on the other hand, when your body had already seroconverted on its own ut the virus found away to mutate and activate itself then a treatment may in needed . you are aldready hbeag negative so you need to ask yourself what the reason for treatment is.. e postives want to turn negative but you're already negative .. so there's no end to treatment. you will have to go on it for life.
its a little hard to be in your shoes now.. you are above the 2000 IU/ml limit for no treatment but you're not shooting up in the millions either so you have a decision to make. if i were you and thats a only a personal opinion, i would monitor for a little bit of time.. one year mayb ...even two years.. 2 years still arent big of a time for monitoring considering if you choose to go with meds then its a life time commitment.. monitor closely for a year or two..maybe once every 3 months to see th elevel of your DNA.. if it goes lower then maybe you'll be ok without meds.. if you goes higher then maybe you needto settle with rlity an go on meds .
up to you and as i always say doctors know better.. but i would recommend you make an appointment with a hepatologist for a second opinion.. they are much more expensive but they do know one or two more things than a gastronologist..
good luck
I know. I just feel like there's sometihing I should be doing even now. Hate waiting to see fit'll get worse. I've read about the homeopathy ans allopathy medicines. herbals supplements etc. I just don't know where to start. Are there any practitioners in the US?
but looks like 'just wait and see' at this point, hsag is eag- and Alt normal. Virus DNA 1,300 IU/ml
--Right.
--Right.
PS: High rheumatoid tither and high cholestrol (sometimes) due to liver inflamation
I have read many of the posts in this forum, and feel like I should give a little history re: my experience so far:
It seems I contracted the infection 2 years ago, I went to my former doctor complaining of body aches. He ordered some blood work and diagnosed rheumatoid arthritis and high cholestrol. He prescribed prednisone for the arthritis and prevastating for the high cholestrol and was on the meds from May of 2007 till 2008. I changed to a new insurance company in 2008 and informed new primary doctor of the diagnosis and meds I was taking. The new insurance required health assesment tests was ordered by primary physician. When I asked for prescriptions for the meds I was on ( old prescription just expired). Because I have no family history of RA, she referred me to a rheumatologist 2 weeks a go, The first question the rheum asked was if I had hep b, I said no. she ordered RA tests and hep panel "just to make sure" she said. Turns out no RA, but yes Hep B. Apparently the RA tither is high for liver inflamation as well. Old doc should have ordered more tests to confirm a definitive RA diagnosis.The Prenisone prescribed to control the RA compromised the body's ability to fight off the hep b. I must have been in the acute hep b infection state when predsone was prescribed. Double bummer!!!. Fortunately the e ag had seroconverted on its own limiting the replication of the virus, hence the low virus count, body may have well been on its way of clearing off the virus!!!, Primary doctor has referred me to a gastroenterologist in 2 weeks but looks like 'just wait and see' at this point, hsag is eag- and Alt normal. Virus DNA 1,300 IU/ml
It seems I contracted the infection 2 years ago, I went to my former doctor complaining of body aches. He ordered some blood work and diagnosed rheumatoid arthritis and high cholestrol. He prescribed prednisone for the arthritis and prevastating for the high cholestrol and was on the meds from May of 2007 till 2008. I changed to a new insurance company in 2008 and informed new primary doctor of the diagnosis and meds I was taking. The new insurance required health assesment tests was ordered by primary physician. When I asked for prescriptions for the meds I was on ( old prescription just expired). Because I have no family history of RA, she referred me to a rheumatologist 2 weeks a go, The first question the rheum asked was if I had hep b, I said no. she ordered RA tests and hep panel "just to make sure" she said. Turns out no RA, but yes Hep B. Apparently the RA tither is high for liver inflamation as well. Old doc should have ordered more tests to confirm a definitive RA diagnosis.The Prenisone prescribed to control the RA compromised the body's ability to fight off the hep b. I must have been in the acute hep b infection state when predsone was prescribed. Double bummer!!!. Fortunately the e ag had seroconverted on its own limiting the replication of the virus, hence the low virus count, body may have well been on its way of clearing off the virus!!!, Primary doctor has referred me to a gastroenterologist in 2 weeks but looks like 'just wait and see' at this point, hsag is eag- and Alt normal. Virus DNA 1,300 IU/ml
Primary doctor said virus load may be too low to begin treatment. Appt scheduled with GI.Any advise?
what other alternatives are there?
--Do not know enough or have not evaluated enough to comment yet. Will post one soon.
--Do not know enough or have not evaluated enough to comment yet. Will post one soon.
By 2007 Guideline, your result is borderline. If you choose treatment of Western medicine, you have the options of antiviral drugs or IFN.
Does the presence of hbe antibody and medium dna count normal ALT and AST stats mean inactive?. Found out about being carrier last Friday. Need to know what to ask my doctor. I a follow up appointment scheduled for tommorow.
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