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Squashing Hepatitis C with no interferon-ribavarin

Hi,

I am a 32 year Hep C survivor. I found and tried a new combo of drugs and supplements that reduced my count from over 500K to 11K in a very short time.

I posted the whole nine yards on Blogger ******* under this lnk    http://******************.********.com/

Not sure if links are allowed.

And no, I am not selling anything. I just want to try and help anyone who is looking for an alternative. Everything I am taking you can easily obtain yourself.

I hope this helps some of you.
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233616 tn?1312787196
james, well yes it was called non-a, non b earlier..which meant they had no idea what it was.  NOn-a non-b is just polite double speak for we are doctors but have no idea what virus this is. It wasn't until we identified it that we can really call it hcv.

they weren't able to identify AND prove by testing for the virus and its genotype until 91-92.

portann,
well that was my point exactly, that there's so much more than meets the eye.
now we add carcinoma as a result of high alt, well makes sense, high alt means more oxidation...more good news...rolleyes
gee, how about this, remember when they showed ants farming aphids for sugars?? yes the ants keep colonies of aphids and such the juices out of them, while leaving them alive...well it turns out these virons do the same, they suc.k out our juices, they farm our lipids and wait for them to harden so they can wear these raincoats out into the bloodstream before returning to their new home, another liver cell.
They also try to change the membrane permeability between liver cells, and when successful they can move from cell to cell without going for that swim....these are some smart little buggers. The more I learn about them, the more I know that very little is not effected in some way shape or form by all their mischief.

Trin and Trish,
I'm just relieved to see we are all willing to think outside the box sometimes. It's in our own interest to be as aware as we can, and also the more that is learned about how this disease behaves, the more doctors find the connections, the more they fear worldwide infection, the more chance of a cure. Its bad enough there are over a billion people with hepatitis, and still the word epidemic is never used.
Don't you wonder though why in all the annals of American medicine not one doctor has ever published a retraction when it is finally proven that his work was in error???

I just think its short sighted to think VL doesn't matter when it does effect all kinds of things, including cell death, meaning alt elevation, which isn't good for myriad reason.

Nobody is trying for king of the hill here, I think we've all made valid points.
But I'm reminded that I was told for years by a doctor that my elevated liver enzymes were no big deal, when in fact they are a big deal, and I had a serious disease.
Many in here have experienced the same.
Which means that sometimes the medical community is wrong, sometimes they don't know all the facts, sometimes it takes them years and years to see correlations.

One reason I think the research concerning VL vs. imflammation is flawed is because they fail to take into account 2 important sub groups. One group is hemophilia, when one has this the immune system is more compromised and hcv can take a worse toll, the other group is half the human race, women. Men and women to be more precise.
SHow me a study which showed the decine of women vs. the decline of men that proves VL has no bearing on health.

Why this is important is because womens organs are smaller than mens, smaller livers by 1/3, but smaller everything generally speaking. That means that we have less ability to recover from the onslaught than men do....and yet what study distinguishes between us?
I'd be curious as to the average lifespan of women vs. men at any given VL. Maybe that would tell a different tale, and one worthy of note perhaps.
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Avatar universal
This is from 2009 and gives a brief overview of some of the the studies out there:

[...While there are many studies regarding the correlation of liver damage with serum HCV-RNA titer, ALT level, and HCV genotypes, the conclusions are quite different. Adinolfi and colleagues held that serum HCV-RNA titer was correlated to the severity of liver damage, which could be accelerated by high HCV load and fat degeneration,6 while Zechini and colleagues suggested the serum ALT level, especially the AST level, was associated with liver damage.7 Puoti and colleagues argued the severity of liver damage and the clinical features had no correlations with HCV load, ALT level, and HCV genotype.8–,10...]

http://labmed.ascpjournals.org/content/40/3/167.full

Whatever the conclusions of this study, it acknowledges in its introduction that there is disagreement among scientists.
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Avatar universal
Speaking for myself, I am not playing a game of "King of the Hill" nor am I unnerved or uncomfortable with new information.  I welcome any data whether it proves or disproves the correlation between viral load and liver health.

I base the informaton I post regarding no correlation between viral load and fibrosis progression on what has been studied, investigated and documented over the last 20 years. This is not to say that the current information is absolute and that it's inconceivable to think that a low viral load plays a role in slowing down the progression of fibrosis. When and if new information arises that negates what has been studied and peer reviewed I will concede my views are incorrect but until those facts are available and wide spread I once again say I stand by the current established data.

Trinity    
Helpful - 0
Avatar universal
Frankly, new information comes to light every day.  I don't think it's a worthless endeavour at all for someone to decide to bring down their viral load, particularly if they have undergone treatment and it has been unsuccessful.  It is far too unknown what influence the viral load DOES have on so many other organisms in the body.  Not enough studies, not enough data for a virus that is relatively new and so much studies yet to be done and new information coming out all the time.  What we know today is far different than what was known five years ago and so on.  Any information people have should be welcome here without it turning into a game of King of the Hill.  I'm finding it interesting to read posts such as Cory's and Bill's and Susan/Portann's and other things.  Like so many other topics, some new information might make people a little uncomfortable at first and be a bit unnerving but far better to be open to new information and adjust approach where necessary to successfully tackle this thing.
Helpful - 0
Avatar universal
Hi Trin,

I hope and guess you're doing great, given your strength and determination.

I wish you well with all my might and rapid success on your next tx. Yay, STAT-C's!

I saw your input tonight about high and low viral load and wanted to point out this very recent and interesting study (2010):

http://jco.ascopubs.org/content/28/30/4587.abstract

"Conclusion Elevated serum levels of HCV RNA and ALT and HCV genotype 1 infection are independent risk predictors of hepatocellular carcinoma. These findings have strong implications for the management of chronic HCV."

Surprising, given the commonly held views we've shared about this...something I'd like to hear from others (such as willing) about in terms of its relevance and/or credibility.

Regards and good night,

Susan (the real portann)

Helpful - 0
1491755 tn?1333201362
The virus was first identified in 1994 ?  Huh didn't know.

In the mid 1970s, Harvey J. Alter, Chief of the Infectious Disease Section in the Department of Transfusion Medicine at the National Institutes of Health, and his research team demonstrated how most posttransfusion hepatitis cases were not due to hepatitis A or B viruses. Despite this discovery, international research efforts to identify the virus, initially called non-A, non-B hepatitis (NANBH), failed for the next decade. In 1987, Michael Houghton, Qui-Lim Choo, and George Kuo at Chiron Corporation, collaborating with Dr. D.W. Bradley from CDC, used a novel molecular cloning approach to identify the unknown organism and develop a diagnostic test.[61] In 1988, the virus was confirmed by Alter by verifying its presence in a panel of NANBH specimens. In April 1989, the discovery of the virus, renamed hepatitis C virus (HCV), was published in two articles in the journal Science.[62][63] The discovery led to significant improvements in diagnosis and improved antiviral treatment.[61]
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