Hello, when I was 23 I experienced high blood pressure and sudden squeezing sensations, and a stopped heartbeat, which then started to race in my chest This occured after using my inhaler more than usual and then exerting myself. I went to the doctor and bp was 123 over 90 heart rate 101, and I felt like someone was squeezing my chest when I laid down. But there were no abnormalities on my ecg, even though I had ingested caffeine.
This evolved into what are now frequent pvcs. They are worse (of course) with caffeine). I did two stress tests, one with caffeine in my system showed non specific st abnormalities with possible anteroseptal infarct (even with caffeine use in the past, I did not have this on my ecg, months before that)
I experienced pvcs during peak exercise. The next stress test was with doppler echo. I was told by the technician that I have systolic anterior leaflet mitral valve prolapse (minimal) with trace regurgitation. But my doctors have told me I am normal normal normal. I am wondering what has changed, something did. I had an aching sensation when I breathed in for months after this initial episode.
now lately, the pvcs that I have been diagnosed with since the fall of 2010 have felt less pounding, softer but still take my breath away, they happened in a store today, and it felt like 10 seconds of continuous pvcs, during which I felt dizzy. I am wondering how one doctor can say I have mitral regurgitation from prolapse and one can say it is just physiological. It is different than it was before and I just want to understand my issue.