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Hashimoto's and Muscle tightness
I have been diagnosed with Hashimoto's. My thyroid levels (T4) are within the normal range even though it went from 0.95 last Nov. to 0.82 this March. I have had severe muscle tightness in my neck and shoulders for the past couple of years and it is only getting worse. I have read about muscle cramps being one of the symptoms and was wondering I am the only person out there that is experiancing this. The muscle on the top of my right shoulder feels like a rock and spasms all the time. Please tell me what I can do for this. My Dr. thinks I'm up in the night and has not put me on any meds for thyroid because the levels are still normal.
I was diagnosed with Hashimoto's 3 weeks ago. I was diagnosed with Hypothyroid about 3 years ago. 15 years ago I was told I had Lupus as I had autoimmune antibodies present in my blood. After more test my primary care doctor said I didn't have Lupus, but they didn't look any further. I've always had a stiff neck and both shoulders are always tight and painful. Up until now I thought it was just me, over stressed.
It's sad because now finding out all the damage that's caused to your body from having a bad thyroid. I was 23 years old and didn't know better to tell my doctor to find out what those antibodies were. I've got degenerative arthritis in my spine, enlarged heart, awful muscle aches and no energy to save my life.
Thanks for listening.
I hope you all feel better soon!!
It's sad because now finding out all the damage that's caused to your body from having a bad thyroid. I was 23 years old and didn't know better to tell my doctor to find out what those antibodies were. I've got degenerative arthritis in my spine, enlarged heart, awful muscle aches and no energy to save my life.
Thanks for listening.
I hope you all feel better soon!!
I too have hard, painful muscles around shoulder and neck area...is there help???
Susan
Susan
I too have Hashimotos and take thyroid hormone (100mg) I am also having neck pain and shoulder paid and very hard muscles between shoulders and neck with some burning pain as well...has anyone out there found relief? Percocet helps me...but I don't want to take this too often...heat rubs and pads I warm in the microwave help but I am only 49 and have a life to live...I do know anxiety doesn't help...what is making our muscles rigid? I have been told its fibromyalgia...and to exercise...which seems to make it worse-seems to be the common soulution...what is the source of the rigid muscles that cause pain and tightness? I want my life to be better...I want to write letters and paint and garden...I am feeling so frustrated.
Susan
Susan
Re the fibromyalgia, the doctor I am seeing next week and lots of thyroid forum members say that 'Fibromyalgia' is just a new name for low thyroid as it only 'appeared' when the stupid TSH test came in and people stopped being treated for thyroid promlems by symptoms al la Dr Barnes. If you look at the symptom lists they are virtually identical.
Lots of poeple on the celiac forums (celiacs also have a high rate of thyroid problems especially Hashis), also have a DX of fibromyalgia, far too many for it to be coincindence, it was this that really made me decide that my GP was totally clueless.
Lots of poeple on the celiac forums (celiacs also have a high rate of thyroid problems especially Hashis), also have a DX of fibromyalgia, far too many for it to be coincindence, it was this that really made me decide that my GP was totally clueless.
My B12 was 415, which my GP said was normal but I later found out that it can cause problems under 500. Honestly is there a reference range for ANY lab test that isn't skewed? LOL I have been taking supps but am not absorbing them. I'v just started seeing a nutritionalist familiar with celiac/thyroid/adrenal (he is sending some adrenal support) and doing testing for exactly what I am absorbing and any yeast/fungus etc that he suspects has moved into my less than perfect digestive tract!
I am also seeing a new Doctor privately next week who treats thyroid, adrenals, the lot and is reccomended by many patient groups in the UK.
I will re read your post when I'v got my thinking head on. a lot to take in and very thought provoking. I have had back/neck for about 16 years, it only got really bad 24/7 2 years or so ago when I had a very stressful job. I think it was only because I have insurance that I actually got anything investigated at all although an actual cause was never mentioned or looked for once I got the 'Fibro' label.
Doctors never seem to look for the cause of anything, just medicate the symptoms.
I 'diagnosed' a dinner guest as celiac over christmas dinner, in about 5 mins flat, yesterday her (private) GI confimed this by biopsy. Poor woman had been ill for years with no cause found and a long list of classic symptoms.
I just want my life back!
I am also seeing a new Doctor privately next week who treats thyroid, adrenals, the lot and is reccomended by many patient groups in the UK.
I will re read your post when I'v got my thinking head on. a lot to take in and very thought provoking. I have had back/neck for about 16 years, it only got really bad 24/7 2 years or so ago when I had a very stressful job. I think it was only because I have insurance that I actually got anything investigated at all although an actual cause was never mentioned or looked for once I got the 'Fibro' label.
Doctors never seem to look for the cause of anything, just medicate the symptoms.
I 'diagnosed' a dinner guest as celiac over christmas dinner, in about 5 mins flat, yesterday her (private) GI confimed this by biopsy. Poor woman had been ill for years with no cause found and a long list of classic symptoms.
I just want my life back!
Have you had your B-12 level checked. It is common to have B-12 definiency with gluten intolerence. Many of your symptoms math as it primarily causeds neuro-psyhiatric problems.
Lack of vitamin B12 produces hematologic abnormalities (megaloblastic anemia) and neurologic complications (subacute combined degeneration of the spinal cord-SCD). The hematologic abnormalities are probably due to a disorder of DNA synthesis. Megaloblastic anemia can be corrected by administration of folate alone. The neuropathological abnormalities are due to a different, as yet unknown, biochemical mechanism. Inherited metabolic disorders of cobalamin-dependent enzymes do not cause SCD. It has been suggested that cobalamin plays a role in the synthesis of cytokines and neurotrophic factors and that dysregulation of this process in cobalamin deficiency damages the white matter.
SCD causes weakness and paresthesias of the distal lower extremities. As the disease progresses, weakness and spasticity of the limbs appear and postural sensation is lost, resulting in unsteadiness of gait. The advanced state is characterized by spastic paraplegia, contractures, ataxia, and impairment of other sensory modalities.
Subacute combined degeneration
The earliest neuropathologic lesion is distention of myelin sheaths, imparting a spongy appearance to the affected white matter. This is followed by disintegration of myelin which is removed by macrophages. Loss of axons also occurs, but is less severe than the loss of myelin. The lesions affect initially the posterior and lateral (combined) columns of the upper thoracic and low cervical spinal cord. They do not affect anatomical fiber systems, but rather involve the white matter in a symmetric nonselective fashion. In advanced cases, the entire circumference of the spinal cord is affected. The optic nerves are rarely involved. All these changes can be prevented (and in their early stages, reversed) by vitamin B12 administration.
Lack of vitamin B12 produces hematologic abnormalities (megaloblastic anemia) and neurologic complications (subacute combined degeneration of the spinal cord-SCD). The hematologic abnormalities are probably due to a disorder of DNA synthesis. Megaloblastic anemia can be corrected by administration of folate alone. The neuropathological abnormalities are due to a different, as yet unknown, biochemical mechanism. Inherited metabolic disorders of cobalamin-dependent enzymes do not cause SCD. It has been suggested that cobalamin plays a role in the synthesis of cytokines and neurotrophic factors and that dysregulation of this process in cobalamin deficiency damages the white matter.
SCD causes weakness and paresthesias of the distal lower extremities. As the disease progresses, weakness and spasticity of the limbs appear and postural sensation is lost, resulting in unsteadiness of gait. The advanced state is characterized by spastic paraplegia, contractures, ataxia, and impairment of other sensory modalities.
Subacute combined degeneration
The earliest neuropathologic lesion is distention of myelin sheaths, imparting a spongy appearance to the affected white matter. This is followed by disintegration of myelin which is removed by macrophages. Loss of axons also occurs, but is less severe than the loss of myelin. The lesions affect initially the posterior and lateral (combined) columns of the upper thoracic and low cervical spinal cord. They do not affect anatomical fiber systems, but rather involve the white matter in a symmetric nonselective fashion. In advanced cases, the entire circumference of the spinal cord is affected. The optic nerves are rarely involved. All these changes can be prevented (and in their early stages, reversed) by vitamin B12 administration.
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