I had my brain MRI on Monday morning and just received my MRI report. Would like opinions if you care to share.
Findings:
Slight encroachment by the cerebellar tonsils on the foramen magnum is accompanied by capsulosynovial proliferative change of the atlanto-odontoid articulation. This constellation of findings constitutes so called Chiari 0 anatomy and has been associated with headaches and neurological symptoms associated with interference of normal CSF migration through the foramen magnum related to cardiac and respiratory activity. No evidence of wedging of the cerebellar vermis, cervico-medullary kinking, syringohydromyelia, cervico-occipital assimialation, or hydrocephalous to suggest High Grade Chiari malformation. Mild asymmetric prominence of the temporal horn of the body of the right lateral ventricle without abnormal signal of the adjacent mesial temporal brain parenchyma.
Periventricular White Matter:
Mild uniform periventricular T2 and FLAIR hyperintensity noted otherwise, without extensive deep or subcortical white matter ischemic gliotic changes, but with accompanying altered signal involving brainstem.
Deep and subcortical white matter:
Solitary punctate focus of T2 and FLAIR hyperintensity within the deep white matter of the right temporal lobe. No perilesional edema, mass effect, thus, consistent with chronic gliosis as a etiology for this white matter hyperintensity in this patient with history of multiple sclerosis.
Brainstem:
Moderate gliosis of the brainstem for which sequela of chronic vertebrobasilar insufficiency is favored in this patient with ectasia of the basilar artery.
Intracranial Vasculature:
Left vertebral dominant, ectatic vertebrobasilar system noted. Evidence of vascular loops in the distribution of the exit root zones of adjacent nerves, without definite neurovascular conflict by artery or vein, thus favoring innocent juxta position of the nerves and vessels. Otherwixe expected flow void signal seen within the major contributors to and tributaries of the circle of Willis as well as the dominant venous outflow tracts.
Evidence of vascular loops in the distribution of the exit root zones of adjacent nerves, without definite neurovascular conflict by artery or vein, for which differential lies between innocent juxta position of the nerves and vessels versus vascular loop syndrome.
Anything you might have knowledge of here would help me. It all makes me wonder about the CCSVI or whatever the initials are. Thanks!!!
Deborah